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- Moreover, a component of endothelial dysfunction has been proposed, which may contribute to abnormal vessel reactivity.
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and recommendations for monitoring. Testosterone substitute in hypogonadal males with angina improves ischaemic threshold and quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone enhance human platelet thromboxane A2 receptor density and aggregation responses.
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Erythrocytosis has been reported as a potential impact of testosterone, which can be helpful in sufferers with anemia. The greatest risk of erythrocytosis occurs with injectable forms of testosterone in contrast with transdermal preparations, especially in supraphysiologic doses. An increase of up to 44% of the baseline hematocrit has been reported .
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We report a case of an acute ST-segment elevation myocardial infarction in a affected person with traditional cardiac danger factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person additionally had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent successful percutaneous intervention of the best coronary artery. Physicians and sufferers ought to pay consideration to the medical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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